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  • Fatty Acid Oxidation Mediated by Malonyl-CoA Decarboxylase Represses . . .
    Fatty acid metabolism reprogramming is a prominent feature of clear cell renal cell carcinoma (ccRCC) Increased lipid storage supports ccRCC progression, highlighting the importance of understanding the molecular mechanisms driving altered fatty acid synthesis in tumors Here, we identified that malonyl-CoA decarboxylase (MLYCD), a key regulator of fatty acid anabolism, was downregulated in
  • Collection - Data from Fatty Acid Oxidation Mediated by Malonyl-CoA . . .
    AbstractFatty acid metabolism reprogramming is a prominent feature of clear cell renal cell carcinoma (ccRCC) Increased lipid storage supports ccRCC progression, highlighting the importance of understanding the molecular mechanisms driving altered fatty acid synthesis in tumors Here, we identified that malonyl-CoA decarboxylase (MLYCD), a key regulator of fatty acid anabolism, was
  • MLYCD mediated Fatty Acids Anabolism Represses Renal Cell Carcinoma . . .
    To investigate the metabolism of clear cell renal cell carcinoma (ccRCC) and elucidate the effect of malonyl-CoA decarboxylase (MLYCD) on fatty acid (FA) anabolism in ccRCC Experimental Design: FA anabolic reprogramming in ccRCC was evaluated in the public database MLYCD expression was measured in clinical samples, cell lines, and Sunitinib-resistant ccRCC cell lines from the mouse model
  • CAN-23-0969 3920. . 3939 - ResearchGate
    ABSTRACT Fatty acid metabolism reprogramming is a prominent feature of clear cell renal cell carcinoma (ccRCC) Increased lipid storage supports ccRCC progression, highlighting the impor-tance of
  • Fatty Acid Oxidation Mediated by Malonyl-CoA Decarboxylase Represses . . .
    Activating MLYCD-mediated fatty acid metabolism could be a promising therapeutic strategy for treating ccRCC MLYCD deficiency facilitates fatty acid synthesis and lipid droplet accumulation to drive progression of renal cell carcinoma, indicating inducing MYLCD as a potential approach to reprogram fatty acid metabolism in kidney cancer
  • National Center for Biotechnology Information
    PMID- 37729394 OWN - NLM STAT- MEDLINE DCOM- 20231204 LR - 20240209 IS - 1538-7445 (Electronic) IS - 0008-5472 (Print) IS - 0008-5472 (Linking) VI - 83 IP - 23 DP - 2023 Dec 1 TI - Fatty Acid Oxidation Mediated by Malonyl-CoA Decarboxylase Represses Renal Cell Carcinoma Progression PG - 3920-3939 LID - 10 1158 0008-5472 CAN-23-0969 [doi] AB - Fatty acid metabolism reprogramming is a prominent
  • Fatty Acid Oxidation Mediated by Malonyl-CoA Decarboxylase . . . - scite
    Supporting: 1, Mentioning: 11 - Fatty acid metabolism reprogramming is a prominent feature of clear cell renal cell carcinoma (ccRCC) Increased lipid storage supports ccRCC progression, highlighting the importance of understanding the molecular mechanisms driving altered fatty acid synthesis in tumors Here, we identified that malonyl-CoA decarboxylase (MLYCD), a key regulator of fatty acid
  • Fatty Acid Oxidation Mediated by Malonyl-CoA Decarboxylase Represses . . .
    Abstract <jats:sec> <jats:title > Fatty acid metabolism reprogramming is a prominent feature of clear cell renal cell carcinoma (ccRCC) Increased lipid storage supports ccRCC progression, highlighting the importance of understanding the molecular mechanisms driving altered fatty acid synthesis in tumors Here, we identified that malonyl-CoA decarboxylase (MLYCD), a key regulator of fatty
  • 郑州大学第一附属医院顾朝辉 周利杰科研团队在国际知名期刊发表重要研究成果-郑州大学医学院
    研究成果以题为“Fatty acid oxidation mediated by malonyl-CoA decarboxylase represses renal cell carcinoma progression”的研究论文发表在国际知名肿瘤学期刊《Cancer Research》上。 该研究发现脂肪酸合成代谢的关键调节因子丙二酰CoA脱羧酶 ( MLYCD )在肾透明细胞癌中表达水平明显降低，
  • Fatty acid metabolism reprogramming in ccRCC: mechanisms and . . . - Nature
    Altered lipid metabolism is a visually obvious, distinguishing feature of clear-cell renal cell carcinoma In this Perspective the authors describe the molecular mechanisms driving fatty acid





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